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Cardiomyocytes in CAD patients exhibit particular vulnerability to ferroptosis under adverse conditions such as ischemia and hypoxia, leading to cellular dysfunction and compromised cardiac performance ( However, research on ferroptosis mechanisms in CHD remains in its nascent stages, with numerous crucial questions awaiting resolution

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Intriguingly, this effect was observed exclusively in female mice, a result that was unexpected because females generally exhibit a more proficient defense against ROS than males [9]

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